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Although the relative contributions of these parameters to the pathophysiology of type 2 diabetes have been debated extensively, it is well established that a feedback loop exists between insulin sensitive peripheral organs and the pancreatic beta-cell [ 2 ]. Insulin resistance in peripheral tissues and pancreatic beta-cell dysfunction are key factors in the development of type 2 diabetes. About · Careers · Privacy · Terms · Contact. Detailed Metabolic Phenotyping in Obesity: Paving the Way Forward BMI The BMI, which is an anthropometric index that is used as a surrogate marker for fat mass and for classifying obesity, is positively associated with risk factors for cardiovascular and metabolic diseases when BMI is above The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of type 2 diabetes.

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Human adipocytes from the subcutaneous superficial layer have greater adipogenic potential and lower PPAR-gamma DNA methylation levels than deep layer adipocytes.

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The Metabolic Phenotype in Obesity: Fat Mass, Body Fat Distribution, and Adipose Tissue Function

Healthy obese versus unhealthy lean: Submit any pending changes before refreshing this page. Indeed, lipodystrophic mice are severely insulin resistant [ 12 ], and surgical implantation of adipose tissue from healthy mice into lipodystrophic animals reversed insulin resistance in a dose-dependent manner [ 13 ]. Detailed metabolic phenotyping, including tissue-specific profiling, is essential to identify individuals or subgroups at increased risk of developing metabolic diseases, and to optimize prevention and treatment strategies. Moreover, deep abdominal subcutaneous adipose tissue seems to expand disproportionally more than the superficial fat depot with increasing obesity, which predisposed toward whole-body insulin resistance and increased cardiovascular risk independent of other adiposity measures in men [ 34 ]. We cut down some trees to make the forest less dense.

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